한빛사 논문
전남대학교
Park, Seon-Young MD, PhD∗; Kim, Jahae MD, PhD†; Chung, Jin Ook MD, PhD‡; Kim, Kangbin MA§∥; Chung, Dong Jin MD, PhD‡
From the ∗Division of Gastroenterology and Hepatology, Department of Internal Medicine
†Department of Nuclear Medicine
‡Division of Endocrinology and Metabolism, Department of Internal Medicine, Chonnam National University Medical School
§Department of Nuclear Medicine, Chonnam National University Hospital
∥Department of Chemistry, Chonnam National University, Gwangju, South Korea.
S.-Y.P. and J.K. equally contributed to this work.
Correspondence to: Jin Ook Chung, MD, PhD
Abstract
Fulminant type 1 diabetes is a recently recognized diabetes subtype characterized by extremely rapid destruction of the pancreatic beta cells, leading to an absolute deficiency in insulin secretion. Fulminant type 1 diabetes is clinically characterized by the drastic onset of hyperglycemia and ketoacidosis within a few days, as well as near-normal glycated hemoglobin (HbA1c) levels despite remarkable hyperglycemia at initial presentation. A 41-year-old woman diagnosed with fulminant type 1 diabetes underwent 68Ga-FAPI PET/CT, which showed intense FAPI uptake throughout the pancreas, especially in the pancreatic tail. Contrast-enhanced abdominal CT failed to reveal any pancreatic abnormalities. This case indicated that 68Ga-FAPI PET/CT might be useful for evaluating patients with fulminant type 1 diabetes.
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