한빛사 논문
Hyun Leea,b, Don D Sina,c,*
aCentre for Heart Lung Innovation, University of British Columbia, St Paul's Hospital, Vancouver, BC V6Z 1Y6, Canada
bDepartment of Internal Medicine, Hanyang University College of Medicine, Seoul, Republic of Korea
cDivision of Respiratory Medicine, Department of Medicine, University of British Columbia, Vancouver, BC, Canada
*Corresponding author.
Abstract
Chronic obstructive pulmonary disease (COPD) is estimated to affect more than 350 million people globally and is responsible for 3·2 million deaths per year, making it the third leading cause of mortality worldwide. Beyond its direct impact in terms of human suffering, COPD contributes as a comorbidity to other leading causes of mortality, including COVID-19, pneumonia, cardiovascular diseases, stroke, and lung cancer, increasing the risk of these conditions and associated mortality by two to four times. Despite the societal burden posed by COPD, research is poorly funded and the disease is often misunderstood. One reason for this is the fallacy that COPD is self-inflicted (by tobacco smoking), irreversible, progressive, and untreatable. A Series of three papers in The Lancet Respiratory Medicine dispels a number of myths about the nature and origins of COPD, providing a state-of-the-art overview of current evidence for a wide range of genetic and environmental risk factors for COPD, and highlighting a new framework to advance our understanding of COPD pathogenesis and the development of new strategies for treatment and prevention.
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