한빛사 논문
In-Cheol Kim and Seongwook Han*
Division of Cardiology, Department of Internal Medicine, Cardiovascular Center, Keimyung University Dongsan Hospital, Keimyung University School of Medicine, Daegu, Republic of Korea
*Corresponding author.
Abstract
With coronavirus disease 2019 (COVID-19), cardiac injury has attracted attention owing to the risk of mortality and morbidity. The incidence of cardiac injury associated with COVID-19 reaches 7–31%, depending on the patient population and definitions. Possible mechanisms of the cardiac injury are angiotensin-converting enzyme 2- (ACE2) mediated direct myocardial injury, hypoxia-induced injury, microvascular damage, and systemic inflammatory response syndrome. There are cases of acute myocarditis as a cardiac manifestation in COVID-19. Among the possible mechanisms of cardiac injury, ACE2-mediated direct myocardial injury and inflammation are specifically suggested as significant contributors to myocarditis associated with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infections.1,2
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