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'uterus' 에 대한 통합검색결과 32건입니다.

한빛사32건

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Uterus-Mimetic Extracellular Microenvironment for Engineering Female Reproductive System
- Adv. Funct. Mater. 07 November 2024 | https://doi.org/10.1002/adfm.202415149
- 2024.11
- 의약학
Endometrial organoids: a reservoir of functional mitochondria for uterine repair
- Theranostics 2024 Jan 1;14(3):954-972 | https://doi.org/10.7150/thno.90538
- 2024.01
- 의약학
Uterus-Derived Decellularized Extracellular Matrix-Mediated Endometrial Regeneration and Fertility Enhancement
- Adv. Funct. Mater. 17 May 2023 | https://doi.org/10.1002/adfm.202214291
- 2023.06
- 바이오·의료융합
CFP1 governs uterine epigenetic landscapes to intervene in progesterone responses for uterine physiology and suppression of endometriosis
- Nat. Commun. 03 June 2023 | https://doi.org/10.1038/s41467-023-39008-0
- 2023.06
- 김연선(Cincinnati Children’s Hospital Medical Center)
An unanticipated discourse of HB-EGF with VANGL2 signaling during embryo implantation
- Proc. Natl. Acad. Sci. USA 2023 May 16;120(20):e2302937120 | https://doi.org/10.1073/pnas.2302937120
- 2023.05
- 생명과학
Linear Association between Radioactive Iodine Dose and Second Primary Malignancy Risk in Thyroid Cancer
- JNCI-J. Natl. Cancer Inst. 2023 Feb 23;djad040 | https://doi.org/10.1093/jnci/djad040
- 2023.02
- 의약학
- 이한솔(충남대학교, 현 Georgia Institute of Technology)
- 이재영(충남대학교)
Hypoxia-alleviating hemoglobin nanoclusters for sensitizing chemo-photodynamic therapy of cervical cancer
- Chem. Eng. J. Volume 457, 1 Feb 2023, 141224 | https://doi.org/10.1016/j.cej.2022.141224
- 2023.02
- 바이오·의료융합
Melatonin inhibits endometriosis development by disrupting mitochondrial function and regulating tiRNAs
- J. Pineal Res. Volume74, Issue1 Jan 2023 e12842 | https://doi.org/10.1111/jpi.12842
- 2023.01
- 생명과학
Liposome-mediated small RNA delivery to convert the macrophage polarity: A novel therapeutic approach to treat inflammatory uterine disease
- Mol. Ther.-Nucl. Acids Volume 30, 13 Dec 2022, Pages 663-676 | https://doi.org/10.1016/j.omtn.2022.11.018
- 2022.12
- 생명과학
- 성진우(Institute of Molecular Biotechnology, Austrian Academy of Sciences (IMBA))
  Interview
Epiblast inducers capture mouse trophectoderm stem cells in vitro and pattern blastoids for implantation in utero
- Cell Stem Cell Volume 29, Issue 7, 7 July 2022, Pages 1102-1118.e8 | https://doi.org/10.1016/j.stem.2022.06.002
- 2022.07
- 생명과학
- 박영롱(Oregon State University)
Targeted Nanoparticles with High Heating Efficiency for the Treatment of Endometriosis with Systemically Delivered Magnetic Hyperthermia
- Small First published: 17 April 2022 | https://doi.org/10.1002/smll.202107808
- 2022.04
- 허동은(University of Pennsylvania)
A microphysiological model of human trophoblast invasion during implantation
- Nat. Commun. Published 15 March 2022 | https://doi.org/10.1038/s41467-022-28663-4
- 2022.03
- 바이오·의료융합
- 최정민(고려대학교 의과대학, Yale University School of Medicine)
Integrated mutational landscape analysis of uterine leiomyosarcomas
- Proc. Natl. Acad. Sci. USA April 13, 2021, 118 (15) : e2025182118 | https://doi.org/10.1073/pnas.2025182118
- 2021.04
- 의약학
An autophagic deficit in the uterine vessel microenvironment provokes hyperpermeability through deregulated VEGFA, NOS1, and CTNNB1
- Autophagy Published online: 17 Jun 2020 | https://doi.org/10.1080/15548627.2020.1778292
- 2020.06
- 의약학
Loss of HDAC3 results in nonreceptive endometrium and female infertility
- Sci. Transl. Med. 09 Jan 2019: Vol. 11, Issue 474, eaaf7533 | DOI: 10.1126/scitranslmed.aaf7533
- 2019.01
- 의약학
- 박연우(Yale University)
Cis-regulatory evolution of forkhead box O1 (FOXO1), a terminal selector gene for decidual stromal cell identity
- Mol. Biol. Evol. First published online: September 15, 2016 | doi: 10.1093/molbev/msw193
- 2016.09
- 신용현(University of Pittsburgh)
Med12 gain-of-function mutation causes leiomyomas and genomic instability
- J. Clin. Invest. 2015;125(8):3280-3284. (First published July 20, 2015) doi:10.1172/JCI81534
- 2015.07
- 이강민(광주과학기술원)
- 김영준(광주과학기술원)
A Neuronal Pathway that Controls Sperm Ejection and Storage in Female Drosophila
- Curr. Biol. Available online 19 February 2015 | DOI: http://dx.doi.org/10.1016/j.cub.2015.01.050
- 2015.02
- 생명과학
β-Catenin activation contributes to the pathogenesis of adenomyosis through epithelial–mesenchymal transition
- J. Pathol. Volume 231, Issue 2, pages 210–222, October 2013 | DOI: 10.1002/path.4224
- 2013.10
차지연

Cincinnati Children’s Research Foundation

1. Can you please briefly summarize this paper? This manuscript describes five significant observations: (1) genetically predisposed females with uterine deletion of Trp53 are more susceptible to preterm birth if exposed to a mild inflammatory stimulus; (2) under these conditions, preterm birth appears to involve both premature decidual senescence and ovarian luteolysis with a drop in progesterone (P4) levels; (3) targeting premature decidual senescence by inhibiting mTORC1 signaling and compensating the drop in P4 levels by exogenous supplementation rescue preterm birth; (4) decidual senescence with increased mTORC1 and COX2 signaling is also evident in women undergoing preterm birth; and (5) cultured human term decidual cells respond to LPS, P4, and rapamycin, as predicted from mouse experiments. Collectively, our findings show that superimposition of inflammation on genetic predisposition results in high incidence of preterm birth and suggest that combined treatment with low doses of rapamycin and P4 may help reduce the incidence of preterm birth in high-risk women. 2. Please introduce your laboratory, university or organization to bio-researchers in Korea. As a former graduate student in the Physician Scientist Training Program (PSTP) at the University of Cincinnati, I have chosen the lab of S. K. Dey in the Division of Reproductive Sciences at Cincinnati Children's Research Foundation (CCRF). CCRF is the research foundation associated with Cincinnati Children's Hospital Medical Center, which is consistently ranked in the top 3 pediatrics hospitals in the nation only behind Children's Hospital Boston and Children's Hospital of Philadelphia. Dr. Dey is an internationally renowned and highly respected reproductive biologist and was recruited in 2008 from Vanderbilt University to inaugurate a Division of Reproductive Sciences at Cincinnati Children's. He has been continuously funded by the NIH for more than three decades, even simultaneously holding two NIH MERIT
- J. Clin. Invest.
- 2013.09.17