한빛사논문
- 조회414
Young Gyun Ko1 Min Ho Kim1 Joo Yong Park2 Chan Hee Gil3 Tae Soo Kim4 Ji-Yeob Choi2,5,6 Doo Hyun Chung7,8 Hyun Jik Kim3 Dong Young Kim3 Hye Young Kim1,9
1Laboratory of Mucosal Immunology, Department of Biomedical Sciences, Seoul National University College of Medicine, Seoul, Republic of Korea
2Laboratory of Behavioral Systems Epidemiology, Department of Biomedical Sciences, Seoul National University College of Medicine, Seoul, Republic of Korea
3Department of Internal Medicine, Seoul National University Hospital, Seoul, South Korea
4Department of Life Science, Ewha Women University, Seoul, Republic of Korea
5Institute of Health Policy and Management, Seoul National University Medical Research Center, Seoul, Republic of Korea
6Cancer Research Institute, Seoul National University, Seoul, Republic of Korea
7Department of Pathology, Seoul National University College of Medicine, Seoul, Republic of Korea
8Laboratory of Immune Regulation, Department of Biomedical Sciences, Seoul National University College of Medicine, Seoul, Republic of Korea
9Institute of Allergy and Clinical Immunology, Seoul National University Medical Research Center, Seoul, Republic of Korea
Correspondence Dong Young Kim, and Hye Young Kim
Young Gyun Ko and Min Ho Kim contributed equally to this study.
Abstract
To the Editor,
Chronic rhinosinusitis (CRS) is a heterogeneous group of chronic inflammatory diseases of the nasal/paranasal cavities.1 It is frequently classified according to the presence or absence of nasal polyps (NP) (CRSwNP and CRSsNP, respectively). Depending on eosinophil numbers, CRSwNP is further classified as eosinophilic (ECRS) or noneosinophilic (NECRS).2 To date, it is unclear how innate lymphoid cells (ILCs) and the nasal epithelium-derived cytokines contribute to these CRS endotypes.
논문정보
- LETTER
- 2022년 03월 (BRIC 등록일 2022-06-22)
- 국내 연구진
- 의약학 > 면역의학
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